产品名称
抗-Olig1抗体, Chemicon®, from rabbit
biological source
rabbit
conjugate
unconjugated
antibody form
purified antibody
antibody product type
primary antibodies
clone
polyclonal
species reactivity
rat, mouse
manufacturer/tradename
Chemicon®
technique(s)
immunocytochemistry: suitable
immunohistochemistry: suitable
immunoprecipitation (IP): suitable
western blot: suitable
NCBI accession no.
UniProt accession no.
shipped in
wet ice
target post-translational modification
unmodified
Quality Level
Gene Information
rat ... Olig1(60394)
Application
使用经验证可用于IP、WB、ICC & IHC的该抗-Olig1抗体检测Olig1蛋白。
研究子类别
神经 & 胶质标记
神经 & 胶质标记
研究类别
神经科学
神经科学
蛋白质印迹：1：2,500-1：5,000，大鼠和小鼠
免疫组化：1：1,000-1：5,000
免疫细胞化学：1：1,000-1：5,000
免疫沉淀：1：50-1：100
最佳工作稀释度必须由最终用户进行确定。
免疫组化：1：1,000-1：5,000
免疫细胞化学：1：1,000-1：5,000
免疫沉淀：1：50-1：100
最佳工作稀释度必须由最终用户进行确定。
Biochem/physiol Actions
Olig-1。通过蛋白质印迹识别 〜27 kDa Olig-1 蛋白。
Disclaimer
除非我们的产品目录或产品附带的其他公司文档另有说明，否则我们的产品仅供研究使用，不得用于任何其他目的，包括但不限于未经授权的商业用途、体外诊断用途、离体或体内治疗用途或任何类型的消费或应用于人类或动物。
General description
27 kDa
Immunogen
重组Olig-1。
Physical form
在 100 mM 甘氨酸，200 mM Tris，pH 8.0 中的液体。
形式：纯化
Preparation Note
自收到之日起，在 2-8° 以未稀释的等分试样可保存 6 个月。
Legal Information
CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany
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存储类别
12 - Non Combustible Liquids
wgk
WGK 1
flash_point_f
Not applicable
flash_point_c
Not applicable
Till Scheuer et al.
Developmental neurobiology, 79(3), 222-235 (2019-01-24)
In preterm infants, the changes from fetal life to ex-utero conditions often coincide with reduced growth and white matter damage of the cerebellum. The premature increase in arterial oxygen tension caused by preterm birth may dysregulate cerebellar development. In a
Huanjia Gao et al.
Frontiers in neuroscience, 18, 1408205-1408205 (2024-07-26)
Vascular dementia (VaD) is a complex neurodegenerative disorder. We previously found that treatment of VaD in middle-aged male rats subjected to multiple microinfarction (MMI) with AV-001, a Tie2 receptor agonist, significantly improves cognitive function. Age and sex affect the development
Kimberly L P Long et al.
Translational psychiatry, 11(1), 631-631 (2021-12-15)
Individual reactions to traumatic stress vary dramatically, yet the biological basis of this variation remains poorly understood. Recent studies demonstrate the surprising plasticity of oligodendrocytes and myelin with stress and experience, providing a potential mechanism by which trauma induces aberrant
Activation of developmental nuclear fibroblast growth factor receptor 1 signaling and neurogenesis in adult brain by ?7 nicotinic receptor agonist.
Narla, ST; Klejbor, I; Birkaya, B; Lee, YW; Morys, J; Stachowiak, EK; Prokop et al.
Stem Cells Translational Medicine null
Kirill Shumilov et al.
Journal of neuroinflammation, 21(1), 171-171 (2024-07-16)
White matter injury (WMI) is thought to be a major contributor to long-term cognitive dysfunctions after traumatic brain injury (TBI). This damage occurs partly due to apoptotic death of oligodendrocyte lineage cells (OLCs) after the injury, triggered directly by the
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