产品名称
驴抗鸡IgY抗体，Cy3偶联物，吸附物种, Chemicon®, from donkey
biological source
donkey
conjugate
CY3 conjugate
antibody form
F(ab′)2 fragment of affinity isolated antibody
antibody product type
secondary antibodies
clone
polyclonal
species reactivity
chicken
manufacturer/tradename
Chemicon®
technique(s)
immunofluorescence: suitable
shipped in
wet ice
target post-translational modification
unmodified
Quality Level
Application
使用经验证可用于IF的该驴抗鸡IgY抗体（Cy3偶联物，吸附物种）检测驴鸡IgY。
Legal Information
CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany
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hcodes
pcodes
Hazard Classifications
Aquatic Chronic 3
存储类别
11 - Combustible Solids
wgk
WGK 3
Richard J Stanton et al.
The Journal of clinical investigation, 120(9), 3191-3208 (2010-08-04)
Human cytomegalovirus (HCMV) in clinical material cannot replicate efficiently in vitro until it has adapted by mutation. Consequently, wild-type HCMV differ fundamentally from the passaged strains used for research. To generate a genetically intact source of HCMV, we cloned strain
Non-epithelial stem cells and cortical interneuron production in the human ganglionic eminences.
Hansen, DV; Lui, JH; Flandin, P; Yoshikawa, K; Rubenstein, JL; Alvarez-Buylla, A; Kriegstein, AR
Nature Neuroscience null
Ileana O Jelescu et al.
NeuroImage, 256, 119277-119277 (2022-05-07)
Biophysical models of diffusion in white matter have been center-stage over the past two decades and are essentially based on what is now commonly referred to as the "Standard Model" (SM) of non-exchanging anisotropic compartments with Gaussian diffusion. In this
Lea Gabele et al.
Frontiers in cellular neuroscience, 18, 1444876-1444876 (2024-08-22)
Influenza A virus (IAV) infection can increase the risk of neuroinflammation, and subsequent neurodegenerative diseases. Certain IAV strains, such as avian H7N7 subtype, possess neurotropic properties, enabling them to directly invade the brain parenchyma and infect neurons and glia cells.
Guy Bar-Klein et al.
Brain : a journal of neurology, 140(6), 1692-1705 (2017-04-27)
A biomarker that will enable the identification of patients at high-risk for developing post-injury epilepsy is critically required. Microvascular pathology and related blood-brain barrier dysfunction and neuroinflammation were shown to be associated with epileptogenesis after injury. Here we used prospective
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