BCR158
苯并[c]吖啶
BCR®, certified reference material
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关于此项目
经验公式(希尔记法):
C17H11N
化学文摘社编号:
分子量:
229.28
Beilstein:
154999
MDL编号:
UNSPSC代码:
41116107
PubChem化学物质编号:
NACRES:
NA.24
等级
certified reference material
Agency
BCR®
制造商/商品名称
JRC
技术
HPLC: suitable
gas chromatography (GC): suitable
包装形式
neat
储存温度
2-8°C
SMILES字符串
c1ccc2nc3c(ccc4ccccc34)cc2c1
InChI
1S/C17H11N/c1-3-7-15-12(5-1)9-10-14-11-13-6-2-4-8-16(13)18-17(14)15/h1-11H
InChI key
OAPPEBNXKAKQGS-UHFFFAOYSA-N
分析说明
For more information please see:
BCR158
BCR158
法律信息
BCR is a registered trademark of European Commission
警示用语:
Danger
危险声明
危险分类
Acute Tox. 4 Oral - Eye Dam. 1
储存分类代码
11 - Combustible Solids
WGK
WGK 3
闪点(°F)
Not applicable
闪点(°C)
Not applicable
W Levin et al.
Cancer research, 43(10), 4625-4628 (1983-10-01)
Benz[c]acridine (B[c]ACR) and 12 of its derivatives, including the 5 metabolically possible trans-dihydrodiols, the diastereomeric bay-region diol-epoxides, 2 non-bay-region diol-epoxides, and the K-region arene oxide, were tested for tumor-initiating activity on mouse skin. A single topical application of 0.4 to
J Molnar et al.
Anticancer research, 13(1), 263-266 (1993-01-01)
Effect of K- and L- molecular orbital regions on expression of antiplasmid and carcinogenic activity was studied with various benz[c]acridine derivatives, tricyclic compounds (acridine orange, phenothiazines) and dibenzoazepine (imipramine). Antiplasmid compounds showed the out-of-phase of molecular orbital in the L-region.
T Kurihara et al.
Anticancer research, 14(5A), 1811-1822 (1994-09-01)
Resonance energies, circuit resonance energies and bond currents of benz[c]acridines were calculated by Aihara's IRE theory. Consequently, it was shown that these compounds had very stable aromatic characters with positive resonance energies and that the resonance energies per pi-electron values
Benz[c]acridine.
IARC monographs on the evaluation of the carcinogenic risk of chemicals to humans, 32, 129-134 (1983-12-01)
J Molnar et al.
Anticancer research, 13(2), 439-442 (1993-03-01)
Some non-differentiation-induction benzo[a]phenothiazines and mutagenic benz[c]acridines more potently inhibited the mitogen-induced blast transformation of human-peripheral blood lymphocytes than differentiation-induction and non-mutagenic counterparts and phenothiazines. Differential absorption spectrophotometry revealed tight complex formation between these drugs and bacterial endotoxin or mitogens. All
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