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Merck
CN

SAB5300165

单克隆抗 OVA 小鼠抗

clone 3G2E1D9, ascites fluid

别名:

N/A

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关于此项目

NACRES:
NA.41
UNSPSC Code:
12352203
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产品名称

单克隆抗 OVA 小鼠抗, clone 3G2E1D9, ascites fluid

biological source

mouse

conjugate

unconjugated

antibody form

ascites fluid

antibody product type

primary antibodies

clone

3G2E1D9, monoclonal

technique(s)

direct ELISA: 1:10,000
western blot: 1:500-1:2,000

isotype

IgG1

shipped in

wet ice

storage temp.

−20°C

target post-translational modification

unmodified

Quality Level

Disclaimer

除非我们的产品目录或产品附带的其他公司文档另有说明,否则我们的产品仅供研究使用,不得用于任何其他目的,包括但不限于未经授权的商业用途、体外诊断用途、离体或体内治疗用途或任何类型的消费或应用于人类或动物。

General description

卵清蛋白(323-339)是由B细胞表位环绕的蛋清蛋白,被特异性IgE抗体结合。除此之外,它还含有CD4+ T细胞表位。

Immunogen

卵清蛋白
小鼠抗卵清蛋白单克隆抗体

Physical form

含 0.03% 叠氮化钠的腹水。

Biochem/physiol Actions

卵清蛋白(OVA)肽323-339在隔离的BALB/c小鼠体内介导25-35%的T细胞对完整OVA的反应。OVA 323-339被广泛用于测定第II类主要组织相容性复合体(MHC)-肽结合特征和T细胞活化特征。它被广泛用于过敏研究。

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存储类别

10 - Combustible liquids

wgk

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable

法规信息

常规特殊物品
此项目有

历史批次信息供参考:

分析证书(COA)

Lot/Batch Number

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访问文档库

Keigo Kondou et al.
Journal of biological engineering, 13, 77-77 (2019-10-28)
The baculovirus (BV) Autographa californica multiple nuclear polyhedrosis virus has been used in numerous protein expression systems because of its ability to infect insect cells and serves as a useful vaccination vector with several benefits, such as its low clinical
Ingrid S Zeelenberg et al.
Cancer research, 68(4), 1228-1235 (2008-02-19)
Expression of non-self antigens by tumors can induce activation of T cells in vivo, although this activation can lead to either immunity or tolerance. CD8+ T-cell activation can be direct (if the tumor expresses MHC class I molecules) or indirect
Comparison between ovalbumin and ovalbumin peptide 323-339 responses in allergic mice: humoral and cellular aspects.
Sun LZ
Scandinavian Journal of Immunology, 71(5), 329-335 (2010)
Tian-Yu Song et al.
Nature communications, 12(1), 7003-7003 (2021-12-03)
Cancer cells acquire genetic heterogeneity to escape from immune surveillance during tumor evolution, but a systematic approach to distinguish driver from passenger mutations is lacking. Here we investigate the impact of different immune pressure on tumor clonal dynamics and immune

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